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Abstract

A comparison was made of the extent of UVR of gamma ray induced lethal damage (mitigation of gamma ray-induced lethal effects by appropriate administration of low UV doses) in fish, amphibian, reptile and mammalian tissue culture cell lines. A significant level of UVR was detected in the non-mammalian lines, but the mammalian cells appeared to have lost this ability. Associated mitotic index data is interpreted as supporting the notion that low UV doses, appropriately administered, may aid repair processes in some cells (indirectly) by hindering the antagonistic metabolic processes which convert gamma ray induced lesions to a non-reversible state.

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